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YK Eleven induces muscle cells to make more follistatin (more than the h0rmone DHT does).
With that said, YK Eleven can be as powerful as t@stosterone in terms of muscle tissue strengthening and muscle cell volumisation. However with the added benefit of being a muscle proliferation compound which contributes to the growth of new muscle proteins.
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How It Works
Myostatin (myo = muscle, statin = stop) is a protein found almost exclusively within skeletal muscle. Myostatin limits muscle growth ensuring that muscles do not grow too large.
The Difference
What’s special about this combination is that while the other $ARMs powerfully stimulate androgen receptors to develop new muscle tissues, YK Eleven has the secondary benefit of suppressing myostatin’s actions to stop further muscle fibre development.
The Result
Being an effective and powerful product, Alpha Helix® is truly a biohacking powerhouse that works by creating the environment for exceeding an individual’s innate physical capacity and entering new territories of physical development.
"what our customers say about
yk Eleven"
FAQ
Most frequent questions and answers
No. YK Eleven (Alpha Helix) is classified as a non-steroidal compound however a compound which is an anabolic potentiator called a SARM (selective androgen receptor modulator)
$arms are compounds that bind to the androgen receptor in a selective manner and therefore act selectively on tissues such as muscle, bone, connective tissue and body fat while avoiding binding to the prostate tissues like regular anabolic compounds and work without shut down of normal hormonal function, althought a slight lowering of T can occur while at higher and longer the recommended dosage levels.
YK 1I works great for cutting body fat, clean bulking, body recomposition and improving body weight strength for training types such as calisthenics.
Alpha Helix is excellent for cutting due to blocking myostatin which decreases muscle gain. Therefore even on a calorie restriction one can maintain muscle mass or even continue building.
For males the dosage is 10mg (1ml) daily. For females the dosage is 1/2 of the male dosage so 5mg (0.5ml) daily.
Research lengths usually run for between 6-8 weeks with 8 weeks being the most effective cycle length.
The potential exists to experience testosterone suppression in male researchers. This is very different to testosterone shut-down caused by stero!d use. If Test. suppression is experienced, it is remedied by ceasing to dose the $arm.
When dosed at the recommended daily mg amounts this potential effect can be minimised or completely avoided.
Females will not experience testosterone suppression or virilisation (developing male characteristics) with YK1!
